Page 13 - 南京医科大学学报自然科学版
P. 13
第43卷第3期 王伟民,赵晨卉,倪思琦,等. ZBTB3表达对胶质母细胞瘤细胞增殖和克隆形成的调控[J].
2023年3月 南京医科大学学报(自然科学版),2023,43(3):297-303 ·303 ·
peutic research for glioma therapy[J]. J Neurol,2018, [16] BRATTON M R,DUONG B N,ELLIOTT S,et al. Regula⁃
265(4):741-756 tion of ERalpha⁃mediated transcription of Bcl⁃2 by PI3K⁃
[5] GUSYATINER O,HEGI M E. Glioma epigenetics:from AKT crosstalk:implications for breast cancer cell survival
subclassification to novel treatment options[J]. Semin [J]. Int J Oncol,2010,37(3):541-550
Cancer Biol,2017,51:50-58 [17] WANG B,ZHAO C H,SUN G,et al. IL⁃17 induces the
[6] YE B,LIU B,YANG L,et al. LncKdm2b controls self⁃re⁃ proliferation and migration of glioma cells through the ac⁃
newal of embryonic stem cells via activating expression of tivation of PI3K/Akt1/NF⁃κB⁃p65[J]. Cancer Lett,2019,
transcription factor Zbtb3[J]. EMBO J,2018,37(8): 447:93-104
e97174 [18] MCFARLAND B C,HONG S W,RAJBHANDARI R,et
[7] LIM J H. Zinc finger and BTB domain⁃containing protein al. NF⁃ κB⁃induced IL⁃6 ensures STAT3 activation and
3 is essential for the growth of cancer cells[J]. BMB Rep, tumor aggressiveness in glioblastoma[J]. PLoS One,
2014,47(7):405-410 2013,8(11):78728
[8] JIN X,WANG J,LI Q,et al. Erratum:SPOP targets onco⁃ [19] NIE Z,CHENG D,PAN C,et al. SH3BGRL3,transcribed
genic protein ZBTB3 for destruction to suppress endome⁃ by STAT3,facilitates glioblastoma tumorigenesis by acti⁃
trial cancer[J]. Am J Cancer Res,2021,11(4):1792-1794 vating STAT3 signaling[J]. Biochem Biophys Res Com⁃
[9] ROTWEIN P. Regulation of gene expression by growth mun,2021,556:114-120
hormone[J]. Mol Cell Endocrinol,2020,507:110788 [20] LI C,YANG J,LEI S,et al. SKA3 promotes glioblastoma
[10] PARK J H,PYUN W Y,PARK H W. Cancer metabolism: proliferation and invasion by enhancing the activation of
phenotype,signaling and therapeutic targets[J]. Cells, Wnt/β⁃catenin signaling via modulation of the Akt/GSK⁃
2020,9(10):E2308 3β axis[J]. Brain Res,2021,1765:147500
[11] ASHRAFIZADEH M,MIRZAEI S,HUSHMANDI K,et [21] XU L,CHEN Y,DUTRA⁃CLARKE M,et al. BCL6 pro⁃
al. Therapeutic potential of AMPK signaling targeting in motes glioma and serves as a therapeutic target[J].
lung cancer:advances,challenges and future prospects PNAS,2017,114(15):3981-3986
[J]. Life Sci,2021,278:119649 [22] WANG L,MA J,WANG X,et al. Kaiso(ZBTB33)down⁃
[12] NATSUIZAKA M,OZASA M,DARMANIN S,et al. Syner⁃ regulation by mirna⁃181a inhibits cell proliferation,inva⁃
gistic up⁃regulation of Hexokinase⁃2,glucose transporters sion,and the epithelial⁃mesenchymal transition in glioma
and angiogenic factors in pancreatic cancer cells by glu⁃ cells[J]. Cell Physiol Biochem,2018,48(3):947-958
cose deprivation and hypoxia[J]. Exp Cell Res,2007,313 [23] PARK H U,SUY S,DANNER M,et al. AMP⁃activated
(15):3337-3348 protein kinase promotes human prostate cancer cell
[13] LIU M,ZHANG Z,WANG H,et al. Activation of AMPK growth and survival[J]. Mol Cancer Ther,2009,8(4):
by metformin promotes renal cancer cell proliferation un⁃ 733-741
der glucose deprivation through its interaction with PKM2 [24] RÍOS M,FORETZ M,VIOLLET B,et al. AMPK activa⁃
[J]. Int J Biol Sci,2019,15(3):617-627 tion by oncogenesis is required to maintain cancer cell
[14] LIM E J,KIM S,OH Y,et al. Crosstalk between GBM proliferation in astrocytic tumors[J]. Cancer Res,2013,
cells and mesenchymal stemlike cells promotes the inva⁃ 73(8):2628-2638
siveness of GBM through the C5a/p38/ZEB1 axis[J]. [25] RAJ C R,FAN Q,JANE A,et al. AMP kinase promotes
Neuro Oncol,2020,22(10):1452-1462 glioblastoma bioenergetics and tumour growth[J]. Nat
[15] ZHANG Y,LIAO Y,CHEN C,et al. p38 ⁃ regulated Cell Biol,2018,20(7):823-835
FOXC1 stability is required for colorectal cancer metasta⁃ [收稿日期] 2022-08-09
sis[J]. J Pathol,2020,250(2):217-230 (本文编辑:陈汐敏)